Most drugs aimed at suppressing HIV target proteins lying on the virus itself, but new research suggests that protein on human immune cell may be key to stopping infection. The human host’s immune cells might work better because human cells mutate at much slower rates than does HIV, so the virus would have much less chance of mutating around the drug.
The research at the U.S. National Human Genome Research Institute is still in its early stages, but it “provides a very nice model that you can inhibit a cellular protein and affect HIV replication.
The study findings are published in this week’s edition of the Proceedings of the National Academy of Sciences.
No User Responded In This Post
Leave Your Comments Here...